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Advanced Pathophysiology Internet Resources Power Point Presentations
Products Sold on our sister site CrystalGraphics. Title: Pathophysiology. Description: Most common cause of surgically correctable hypertension. May result from atherosclerosis, fibromuscular dysplasia, or post traumatic Tags: atherosclerosis pathophysiology. Latest Highest Rated. May result from atherosclerosis, fibromuscular dysplasia, or post traumatic subintimal dissections.
Accounts for a disproportionate fraction of hypertension in children, young adults, and new-onset hypertension in the elderly. Often refractory to medical management Pt. Other causes of surgical correctable hypertension include pheochromocytoma, aldosterone-secreting tumors, descending thoracic aortic coarctation.
It is three times more common in women than in men. Usually occurs in the 2nd to 4th decade.
Bilateral involvement is seen in as many as 50 of cases. This change stimulates the renal JGA to produce renin which catalyzes the conversion of angiotensinogen to angiotensin I. Stimulates the production of aldosterone, causing sodium retention increased plasma volume.After you enable Flash, refresh this page and the presentation should play.
Get the plugin now. Toggle navigation. Help Preferences Sign up Log in. To view this presentation, you'll need to allow Flash. Click to allow Flash After you enable Flash, refresh this page and the presentation should play. View by Category Toggle navigation. Products Sold on our sister site CrystalGraphics. Title: Cardiac Pathophysiology. Tags: aneurysm aortic cardiac pathophysiology. Latest Highest Rated. Title: Cardiac Pathophysiology 1 Cardiac Pathophysiology 2 Pericarditis Often local manifestation of another disease May present as Acute pericarditis Pericardial effusion Constrictive pericarditis 3 No Transcript 4 Acute Pericarditis Acute inflammation of the pericardium Cause often unknown, but commonly caused by infection, uremia, neoplasm, myocardial infarction, surgery or trauma.
Membranes become inflamed and roughened, and exudate may develop 5 Symptoms Sudden onset of severe chest pain that becomes worse with respiratory movements and with lying down. Generally felt in the anterior chest, but pain may radiate to the back. May be confused initially with acute myocardial infarction Also report dysphagia difficulties swallowingrestlessness, irritability, anxiety, weakness and malaise 6 Signs Often present with low grade fever and sinus tachycardia Friction rub sandpaper sound may be heard at cardiac apex and left sternal border and is diagnostic for pericarditis but may be intermittent ECG changes reflect inflammatory process through PR segment depression and ST segment elevation.
If development is slow, pericardium can stretch If develops quickly, even 50 ml of fluid can cause problems When pressure in pericardium diastolic pressure, get? Today, usually idiopathic, or associated with radiation exposures, rheumatoid arthritis, uremia, or coronary bypass graft 15 Pathophysiology Fibrous scarring with occasional calcification of pericardium Causes parietal and visceral layers to adhere Pericardium becomes rigid, compressing the heart??
Heart Murmur sound caused by turbulent blood flow through damaged valves. This leads to chamber dilation and hypertrophy. Chamber dilation and myocardial hypertrophy are compensatory mechanisms to increase the pumping capability of the heart. Eventually, the heart fails from overwork 31 Aortic Stenosis Three common causes Rheumatic heart disease -Streptococcus infection damage by bacteria and auto-immune response Congenital malformation Degeneration resulting from calcification 32 Aortic Stenosis Blood flow obstructed from LV into aorta during systole Causes increased work of LV?
LV dilation hypertrophy as compensation? Marfan syndrome or atherosclerosis 38 Reflux of blood from aorta to LV during ventricular relaxation.
LV hypertrophy LV takes on globular shape? Both LV and LA pump harder to move same blood twice? LV hypertrophy and dilation as compensation Compensation works awhile, then see? LA hypertrophy? Studies suggest an autosomal dominant inheritance pattern Many cases completely asymptomatic Regurgitant murmur or midsystolic click 45 No Transcript 46 Clinical manifestations Palpitations Tachycardia Light-headedness, syncope, fatigue, weakness Chest tightness, hyperventilation Anxiety, depression, panic attacks Atypical chest pain 47 Management Echocardiography for diagnosis Related to degree of regurgitation Antibiotics before invasive procedures Beta blockers to relieve syncope, severe chest pain, or palpitations Avoid hypovolemia Surgical repair 48 General Treatment for Valve disorders Antibiotics for Strep Anti-inflammatories for autoimmune disorder Analgesics for pain Restrict physical activity Valve replacement surgery 49 Heart failure Definition When heart as a pump is insufficient to meet the metabolic requirements of tissues.
Acute heart failure 65 survival rate Chronic heart failure Most common cause is ischemic heart disease 50 Right heart failure Systemic symptoms Edema, ascites Enlarged liver, spleen Swollen feet, ankles Nausea Swollen internal jugular veins 51 Left hear failure Fluid accumulation in lungs Shortness of breath Orthopnea Coughing, foaming sometimes Tiredness weakness 52 Ischemic Heart Disease Coronary Artery Disease CADmyocardial ischemia and myocardial infarction are progression of conditions that impair the pumping ability of the heart by depriving it of oxygen and nutrients.
Most common cause is atherosclerosis 54 The arteries that supply the heart are the first branches off the aorta Coronary artery disease decreases the blood flow to the cardiac muscle.
Persistent ischemia or complete occlusion leads to hypoxia. Hypoxia can cause tissue death or infarction, which is a heart attack, which accounts for about one third of all deaths in U. Increased contractility About minutes after blockage, if no relief Cardiac infarct cell death 58 Clinical Manifestations May hear extra, rapid heart sounds ECG changes T wave inversion ST segment depression 59 No Transcript 60 Chest Pain First symptom of those suffering myocardial ischemia.
Often occurs at night during REM sleep May result from hyperactivity of sympathetic nervous system, increased calcium flux in muscle or impaired production of prostaglandin 64 Unstable Angina pectoris Lasts more than 20 minutes at rest, or rapid worsening of a pre-existing angina May indicate a progression to M.
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Hello, I am starting my advanced pathophysiology class and I am kind of freaking out about it. Wondering if anyone would be willing to share some of their notes with me?? I have purchased a supplemental book along with the book for my class but could really use any help that is offered to succeed in this class. Thank you to anyone that is willing to help me. There is no such thing as a 'standardized' course - LOL.
Notes from students in other schools may not be much help to you at all. Do you have a "Guyton"? That was my 'bible' in all my advanced patho courses It's pricey, but you get online access as well. It is frequently updated, so I have no idea which edition is the current one. MY Patho course is nightmarishly difficult, the instructor literally gives us pages of power points each week and expects us to know everything, needless to say I am getting an excellent education but I don't know if I'll live long enough to enjoy the benefits due to the stress.
All whining aside, I have Clinical Pathophysiology made ridiculously simple by Berkowitz, and a textbook, Symptom to Diagnosis, an evidence based guide by Lange. These two books are excellent resources. The patho book condenses everything and has very good diagrams, the Symptom book has many, many case studies for each main diagnosis and walks you through each differential.
I would suggest googling things that don't make sense to you too, I found many great resources on the web that helped me put some of the more difficult concepts together.
Good Luck. Thanks for the advice on materials!! I am hearing the same kind of thing about this class that I will be taking which I am very nervous about!!!
Hope I make it through! Practice review questions on Quizlet and Cram. I passed with an A. A midterm and a Final. It is doable but you will not have a life. I'm starting this August. I'm glad to see that everyone is still alive so far.Anatomy and physiology of the respiratory system
Anyone helpful tips would be very welcome. I will be starting full time, I am in the graduate stats class now. I'm starting Patho this fall also. Would be nice to keep this thread alive and share resources. Thanks for the links and ideas so far. I also will be in patho this fall too.Summer Investigates human responses to stress and disease by analyzing selected clinical cases, comparing study findings with accepted health states, and applying research based diagnostic methods.
In-depth study of a pathophysiologic condition selected by the student enhances integration of concepts learned by presentation, discussion, and literature review. Listen on Apple Podcasts. Extremely valuable supplement to my online DNP Pathophysiology class at a major university. The art of lecturing is alive and well with this professor. Excellent nurse educator and skilled lecturer. Profoundly helpful for taking patho online much more accessible lectures than the voiceover content from my university.
The stories from practice make the content stick. This instructor is absolutely amazing! Thank you so much for this podcast. The only other thing that would make it better is if I could print the PowerPoints. Her lectures are so informative!
Apple Podcasts Preview. Customer Reviews See All. The Peter Attia Drive. Peter Attia, MD. Celine Gounder and Ronald Klain. Mark Hyman. Scott D. Rhonda Patrick, Ph. The Curbsiders Internal Medicine Podcast. Biology -- Pathophysiology with Doc C.
Gerald Cizadlo. Pharmacology for Future Nurses - Content. Brenda Martin. Emergency Medicine Lectures. Allan Forsman. Allan Forsman Health Sciences.Find an article on a genetic disorder and summarize in two or three paragraphs the genetic component causing the disorder and any multifactorial inheritance components that may contribute to the disorder.
Discuss the usual age of disease onset and if the sex-specific threshold model fits the disorder. What education could you present to high-risk patients to reduce the risk of disease onset if a multifactorial component exists?
Share your thoughts on the legal, ethical, and social implications that may be related to genetic screening. How would you educate your patient that is considering having genetic screening?
Address any adverse effects the medication may have based on age, pregnancy, or lactation.
Active acquired immunity can be achieved through the use of childhood immunizations. Many parents voice concerns regarding the safety of vaccinations. What is your opinion on the current use of vaccinations?
How would you educate parents regarding the safety and effectiveness of vaccinations? Defend your answers with evidence-based research. Share a case study of an electrolyte imbalance from your practice or from the literature.
Advanced Pathophysiology Internet Resources Power Point Presentations
Summarize the case study in paragraphs. Then discuss the clinical manifestations of the imbalance, the pathophysiology behind the imbalance, normal cell membrane transport of the electrolyte sand any alterations in cell membrane transport caused by the imbalance.
How was the electrolyte imbalance resolved? Analyze the case study to determine any areas in which patient or staff education may have helped to prevent the electrolyte imbalance. Use at least three scholarly, peer-reviewed resources less than 5 years old in addition to course materials.
A widespread belief is that caffeine has a diuretic effect when consumed and will induce dehydration if used by athletes or those who perform strenuous exercise. Discuss the pathophysiology behind this belief. Share your thoughts on whether you agree or disagree that caffeine can induce dehydration. Support your answer with evidence-based literature. Compare and contrast the pathophysiology between chronic obstructive pulmonary disease COPD and pneumococcal pneumonia.
Include any types of cellular injury or cellular adaptation that may occur. Evaluate if an inflammatory response is present and discuss the impact of that response. Find an evidence-based journal article on cystic fibrosis and summarize the article in one or two paragraphs. Address why you chose the article and how you might use the findings in your current or future practice setting.
Atherosclerosis is a common disorder of the arteries. A modifiable risk factor for the development of atherosclerosis is hypertension.
Pathophysiology - PowerPoint PPT Presentation
Discuss the pathophysiology of both disorders and the mechanisms by which hypertension contributes to atherosclerosis. Discuss the current medications used to treat both disorders and the pharmacologic actions the medications have in altering the pathophysiology. How can you use this information in your current or future practice setting?I carefully chose the very latest I could find — pathophysiology is quite an evolving field! But many of these presentations were never intended for nursing students — so they can be over the top with information that is much more than you need.
In every one of these presentations there are gems to find — the graphics in particular can make a huge difference in helping students understand a concept.
I encourage you to browse them carefully to look for these gems. Basic Pathophysiology slides from Jones and Bartlett that do a great job in providing the basics. I like to provide this resource to help students and to emphasize that there is a difference between basic and advanced patho. Pathophysiology of Diabetes …. Be the Pancreas! Nice job done by med-surg. Pathophysiology of Cancer From Arkansas State University It would be impossible to cover all of the various cancers, but an understanding of the basic pathophysiology is a good way to manage this important content.
This is also increasingly important with growing numbers of people struggling with chronic pain. Pulmonary Case Studies Loyola University. Cardiovascular Pathophysiology McGraw Hill. Home About Dr. A note about these Power Point presentations…… I carefully chose the very latest I could find — pathophysiology is quite an evolving field!This graduate-level course is 14 To enroll, speak with an Enrollment Representative.
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Transferability of credit is at the discretion of the receiving institution. Home Courses HCS Scroll for more. Get Started. Request Info. This course provides students with advanced anatomy, physiology, and pathophysiology of systems in relation to an individual's health across the lifespan. Focus is given to the physiological and biological manifestations and adaptive and maladaptive changes that occur. The knowledge gained in this course lays a foundation for the management of primary care issues of the family and its members.
Course details: Credits: 4. Disorders of the Immune and Musculoskeletal Systems Analyze normal anatomical structures and physiological processes of the musculoskeletal systems. Correlate anatomical structures and physiological mechanisms of the musculoskeletal systems with pathophysiological signs and symptoms of disease. Compare and contrast the characteristics and pathophysiology of select autoimmune and musculoskeletal symptoms with similar symptoms caused by other organ systems or pathophysiological processes.
Analyze the physiology and pathophysiology of select autoimmune and musculoskeletal diseases. Neurologic System Analyze normal anatomical structures and physiological processes of the neurologic system.
Correlate anatomical structures and physiological mechanisms of the neurologic system with pathophysiological signs and symptoms of disease. Compare and contrast the characteristics and pathophysiology of select neurologic symptoms with similar symptoms caused by other organ systems or pathophysiological processes.
Analyze the physiology and pathophysiology of select neurologic diseases. Gynecological and Genitourinary System Compare and contrast the characteristics and pathophysiology of select gynecological and genitourinary symptoms with similar symptoms caused by other organ systems or pathophysiological processes. Analyze the physiology and pathophysiology of select gynecological and genitourinary diseases.